By continuing to use this site you consent to the use of cookies on your device as described in our cookie policy unless you have disabled them. Controlled burst suppression should be present on EEG. Studies have indicated that plasma and CSF levels of pentobarbital do not accurately reflect the physiologic effects of pentobarbital and recommend monitoring the EEG instead of the pentobarbital levels. Cheapest Solution: Meds prescription for rhomboid/shoulder blade pain - Answered by a verified Doctor We use cookies to give you the best possible experience on our website. Once burst suppression is achieved then a constant infusion may be maintained. Treatment is started with a loading dose and maintained with an infusion. EEG monitoring is helpful in defining the limits of therapy by the occurrence of burst suppression. Intensive monitoring including EEG, arterial blood pressure monitoring, PA catheter monitoring and frequent blood chemistries is required for barbiturate coma therapy. The mechanism of action of barbiturates is by inhibiting the synaptic transmission thereby decreasing the cerebral. Barbiturates may also be protective during periods of cerebral hypoxia. In addition to lowering ICP, extracellular concentration of lactate and excitatory amino acids are reduced after barbiturate therapy. Barbiturate coma therapy has been used in head injured patients to lower ICP when other therapies such as mannitol and hyperventilation have failed. The mechanism of action of barbiturates is by inhibiting the synaptic transmission thereby decreasing the cerebral metabolic rate of oxygen consumption, reducing blood volume and ICP. ![]() ![]() Barbiturate coma therapy has been used in head injured patients to lower ICP when other therapies such as mannitol and hyperventilation have failed.
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